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ntration of the three berry extracts;

endothelium-dependent, NO mediated relaxation or direct NO mediated relaxation of coronary arteries is modified following either short term or overnight incubation of the arteries with a physiological concentration of the three berry extracts; and

impaired endothelium-dependent, NO mediated relaxation of coronary arteries following exposure to superoxide is alleviated by concurrent exposure of the arteries to a physiological concentration of the three berry extracts.

Results
They found that:

Extracts from chokeberry and bilberry, but not elderberry, produce a direct dose-dependent relaxation of porcine coronary arteries, with chokeberry extract being the most potent. This relaxation was totally dependent on the vascular endothelium because the extracts did not significantly contract or relax arteries in which the endothelium was removed.

Relaxation of coronary arteries to an agent that releases NO from the arterial endothelium, as well as that in response to NO itself, were not altered by either the presence, short term exposure or overnight exposure to a physiological concentration of any of the extracts.

A physiological concentration of chokeberry extract totally protected the coronary arteries from oxidant injury. Extracts from bilberry and elderberry provided partial, but not complete, protection against oxidant injury.

Conclusions
Berry extracts, in concentrations likely to be seen in the human blood stream following reasonable ingestion of these compounds, can stimulate the coronary arterial NO system. This is potentially important because of the pivotal role this system has in maintaining normal arterial function.

Furthermore, the variation in direct coronary vasoactivity and oxidative protective effect of the three different extracts, which were normalized for total anthocyanin content, strongly suggests that an individual component, or c
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Contact: Donna Krupa
djkrupa1@aol.com
703-967-2751
American Physiological Society
22-Apr-2002


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