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Better model of cancer development sheds light on potential angiogenesis target

osure, the team's preliminary findings suggest that cancers may develop faster in mice without Id1 because inactivation of the Id1 gene triggers alterations in a receptor on skin immune cells called gamma delta T cells. With a faulty receptor, these cells fail to migrate to the skin to fight off cancer cells.

"We realize that studies based on tumor transplant models are quicker and easier to perform in the laboratory, but it's important to study both the transplant and genetic models to get a clear picture of how genes interact," she says. The researchers believe that the tumor transplant model is most similar to the process of cancer metastasis, in which Id1-associated angiogenesis is likely to play an important role.

The research was funded by the National Institutes of Health, the Flight Attendant Medical Research Institute, the American Skin Association, and the V Foundation.

Study participants include Hashmat Sikder, David L. Huso, Binghe Wang, Byungwoo Ryo, and Jonathan D. Powell from Johns Hopkins; Hong Zhang and Sam T. Hwang from the National Cancer Institute.


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Contact: Vanessa Wasta
wastava@jhmi.edu
410-955-1287
Johns Hopkins Medical Institutions
20-Oct-2003


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