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Common genetic damages in non-dividing cells lead to the creation of mutant proteins

to devise explanations for physiological changes that occur in non-dividing cells exposed to damaging environmental agents.

"A number of studies, culminating in this one, show that DNA damages leading to TM are an important event that may account for the deleterious effects of unrepaired genetic damage. Although our study was in e.coli, very similar systems operate to repair genetic damage in human cells, thus this is a very important model for helping understand the mechanisms in non-dividing cells that can cause the manufacture of mutant proteins as a result of genetic damage to cells, says Dr. Doetsch."

Other contributors to the research were Bernard Weiss, PhD, Emory professor of pathology and laboratory medicine, Zara A. Doddridge, PhD, Emory postdoctoral fellow, and Ho Jin You, MD, PhD, from the Department of Pharmacology at Chosun University Medical School in the Republic of Korea.


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Contact: Holly Korschun
hkorsch@emory.edu
404-727-3990
Emory University Health Sciences Center
22-Oct-2003


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