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Cyclis Pharmaceuticals reports studies defining new mechanism for potential cancer therapies

her showed that -lapachone-treated cancer cells, but not normal cells, exhibit elevated E2F1 protein levels, indicating activation of a specific checkpoint apoptosis pathway. Prior published research by Dr. Li and colleagues has demonstrated the effectiveness of -lapachone in animal models.

In addition to Dr. Li, other authors on the paper were Youzhi Li, Ph.D., Xiangao Sun, Ph.D., both of whom are now at Cyclis, and Arthur Pardee, Ph.D., Professor Emeritus at the Dana-Farber Cancer Institute and a member of the Cyclis Scientific Advisory Board.


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Contact: Shannon Altimari
saltimari@fkhealth.com
617-761-6727
Feinstein Kean Healthcare
18-Feb-2003


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