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Findings aid understanding of neurodegenerative diseases

"How do we use this new information about molecular chaperones to our advantage, to protect individuals from the molecular damage of disease?" said Morimoto. "That is our next challenge."

Related to this research, Morimoto reported in the August issue of Proceedings of the National Academy of Sciences that the appearance of aggregates and polyglutamine protein toxicity associated with Huntington's disease in C. elegans can be suppressed by a genetic pathway that controls aging.


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Contact: Megan Fellman
fellman@northwestern.edu
847-491-3115
Northwestern University
29-Sep-2002


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