Genetically modified mice provide information on treatments for cocaine dependence

that had been raised under identical conditions, but did not have genetic deletions of both the dopamine and the norephinephrine transporter gene.

Since inactivation of dopamine and norepinephrine transporters in mice creates a situation that partially mimics cocaine's effects on dopamine and norepinephrine levels in the brain, these observations imply that cocaine has actions beyond these two targets. Researchers in this group have already suggested that the serotonin system may be an additional target for cocaine and these results are consistent with that contention. This research is supported by the National Institute of Drug Abuse.


Contact: Sarah Goodwin
Federation of American Societies for Experimental Biology

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