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Inhibition Of A Novel Gene Involved In Phosphorylating Starch In TransgenicPotato Tubers Leads To The Repression Of Cold Induced Sweetening

t Physiology in Golm-Potsdam have isolated proteins bound to potato starch granules, raised antisera to these proteins and used the antisera to screen cDNA libraries for corresponding clones. One of the resulting clones, designated R1, is encoding a 160 kDa protein that is partially localized on starch granules.

Lorberth et al. describe in Nature Biotechnology (Vol.16 (5) - May 98) the effects on starch metabolism exerted if the expression of this gene is inhibited in transgenic potato plants using antisense technology. A major observation made is the reduction of the phosphate monoester content of the starch synthesized in the transgenic lines down to 10% as compared with wild-type plants. This indicates that the R1 protein is responsible for the phosphorylation of starch, which is also supported by the fact that the expression of the protein in Escherichia coli leads to elevated phosphate contents of the synthesized glycogen. The existance of the R1 protein is not confined to the Solanaceous species; corresponding sequences are also found in Arabidopsis and rice, but not in bacteria, mammals and yeasts, indicating that it is a general but unique component of starch biosynthesis.

The biochemical mechanism by which the R1 protein phosphorylates amylopectin remains to be elucidated. It is not known which substrate acts as a phosphoryl donor, nor which type of glucans are phosphate acceptors. Evidence that phosphoenolpyruvate may act as a phosphoryl donor comes from slight sequence homology of the R1 protein to the PEP synthases from different bacteria. The question of a phosphoryl acceptor is far more difficult to address, as the different intermediates between ADP-glucose and amylope
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Contact: Jens Kossmann
Kossmann@Mpimp-Golm.MPG.de
+49-331-9772-784
Max-Planck-Gesellschaft
14-May-1998


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