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Lymphoma gene found

promotes either cell cycle arrest or cell death. Thus, when Dmp1 is nonfunctional, the p53 pathway is disrupted. By disabling Dmp1, lymphoma cells gain a selective advantage and obviate the selection for Arf deletion or p53 mutation that often occurs during Myc-induced lymphomagenesis.

The identification of additional tumor suppressor genes, like Dmp1, that promote tumorigenesis lends insight into the genetic culprits that may be responsible for tumor development when the list of usual suspects fails.


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Contact: Nora Poppito
poppito@cshl.org
Cold Spring Harbor Laboratory
14-Nov-2001


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