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Researchers discover how herpes tricks the immune system

t helps control latent infection. Using special tests that reactivate latent viruses, the team found that three to five times more virus could be reactivated in C3-deficient mice than in normal mice.

"Our findings explicitly show that complement plays a role during persistent and latent infection, and that was unexpected," says Virgin. "They also emphasize that we can't study a viral protein during just one part of a virus's life cycle and assume we understand the function of that protein. It's important to look at it during all phases of infection."


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Contact: Darrell E. Ward
wardd@msnotes.wustl.edu
314-286-0122
Washington University School of Medicine
20-Aug-2002


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