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JCI table of contents, November 15 2004

ase, proving molecular confirmation of a novel genetic concept.

AUTHOR CONTACT: Jorge Frank

Department of Dermatology and Allergology, University Clinic of the RWTH, Aachen, Germany.

Phone: 49-241-808-9162; Fax: 49-241-808-2413; E-mail: jfrank@ukaachen.de.

View the PDF of this article at: https://www.the-jci.org/press/21791.pdf

ACCOMPANYING COMMENTARY:
TITLE: Piecing together the puzzle of cutaneous mosaicism.

AUTHOR CONTACT: Amy S. Paller
Department of Dermatology. Northwestern University, Chicago, Illinois, USA.
Phone: 312-695-0197; Fax: 312-695-0664; E-mail: apaller@northwestern.edu.

View the PDF of this commentary at: https://www.the-jci.org/press/23580.pdf

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Understanding the development of wasting in disused muscles

For people with immobilized limbs, sedentary lifestyles or subject to extended periods of bed rest, muscle disuse often leads to the loss of muscle tissue known as muscle atrophy. Earlier studies have implicated three proteins NF-kappaB, p50, and Bcl3 in the development of atrophy, however the precise mechanisms involved have remained unknown. In the November 15 issue of the Journal of Clinical Investigation, Susan Kandarian and R. Bridge Hunter from Boston University examined mice deficient in the proteins p105/p50 (encoded by the gene Nfkb1) or Bcl3 (encoded by the gene Bcl3) and had their hind limbs suspended for 10 days. The authors found that these mice lost soleus muscle mass. While normal mice showed greatest loss of their fast contracting muscle fibers in comparison to slow contracting fibers, the fast cont
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Contact: Brooke Grindlinger
press_releases@the-jci.org
212-342-9006
Journal of Clinical Investigation
15-Nov-2004


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