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JCI table of contents November 1, 2004

ibutes to HDL cholesterol in the general population

AUTHOR CONTACT:
Anne Tybjrg-Hansen
Copenhagen University Hospital, Blegdamsvej 9, DK-2100 Copenhagen , Denmark
Phone: 45-3545-4159; Fax: 45-3545-4160; E-mail: at-h@rh.dk

View the PDF of this article at: https://www.the-jci.org/press/20361.pdf

ACCOMPANYING COMMENTARY: Do DNA sequence variants in ABCA1 contribute to HDL cholesterol levels in the general population?

AUTHOR CONTACT:
Pivi Pajukanta
University of California, Los Angeles, 695 Charles E. Young Dr South, Los Angeles, CA 90095, USA
Phone: 310-267-2011; Fax: 310-794-5446; E-mail: ppajukanta@mednet.ucla.edu

View the PDF of this article at: https://www.the-jci.org/press/23466.pdf


Gene that Blocks Free Radical Damage Protects Against Emphysema

Emphysema is a major manifestation of chronic obstructive lung disease, which affects more than 16 million Americans and is the fourth highest cause of death in the United States. This disease is primarily cigarette smoke induced, but oxidative stress, a harmful condition that occurs when there is an excess of free radicals, has recently been alleged to play an important role in lung susceptibility to cigarette smoke-induced damage. To investigate the impact of oxidative stress on emphysema development, Shyam Biswal and colleagues, from Johns Hopkins University, disrupted in mice the nuclear factor, erythroid-derived 2, like 2 (Nrf2) gene, which makes a protein that regulates genes involved in protecting the body from free radical damage. The authors found that cigarette smoke-induced emphysema was of earlier onset and more widespread in the Nrf2/ mice than in their wild-type littermates. The Nrf2/ mice also had a greater num
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Contact: Laurie Goodman
press_releases@the-jci.org
212-342-4159
Journal of Clinical Investigation
1-Nov-2004


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