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JCI table of contents November 1, 2004

time organs and tissues may be harvested as needed and allowed by . The authors propose a similar mechanism might be used for embryonic cells. At this stage, however, no definition for death has been defined for unformed humans. The authors highlight the point that many embryos created for in vitro fertilization are not used because they are unable to properly produce a fetus due to inherent genetic or mechanical defects. These embryos, however, often contain some normal cells that, while unable to create a fully formed human, can be donated for use. The arguments defined by Landry and Zucker are laid out with historical background both on the history of organ donation and in vitro fertilization. The commentary offers a potential path for both sides of the embryonic stem cell argument to follow to common ground in the discussion for determining an ethically and medically sound future for stem cell research.

TITLE: Embryonic death and the creation of human embryonic stem cells

AUTHOR CONTACT:
Donald W. Landry
Columbia University, 630 West 168th St, New York, NY 10032, USA
Phone: 212-305-2436; Fax: 212-305-3475; E-mail: dwl1@columbia.edu

View the PDF of this article at: https://www.the-jci.org/press/23065.pdf


Diabetes Drug Works by Enhancing Fat Cell Energy Production

Energy homeostasis is controlled by a complex series of cellular and hormonal interactions. White fat tissue has been shown by mouse-knockout studies and the identification of fat-specific secreted factors to be central to this process. Drugs for type 2 diabetes that enhance sensitivity to insulin, such as rosiglitazone, work through mechanisms that involve fat. Cell culture work has indicated that rosiglitazone alters the mitochondria of fat cells, both in their structural features and in the types of proteins they produce. Mitoch
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Contact: Laurie Goodman
press_releases@the-jci.org
212-342-4159
Journal of Clinical Investigation
1-Nov-2004


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