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Monell researchers find metabolic defect in liver that can lead to obesity

ight loss in obese rodents.

With this in mind, Friedman notes, The present findings point to fat oxidation in the liver as a target for the development of drugs that suppress appetite and promote weight loss in obese individuals.

Future studies will guide development of such interventions by examining more closely the function and activity of the target enzymes.


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Contact: Leslie Stein
stein@monell.org
267-519-4707
Monell Chemical Senses Center
24-Jul-2007


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