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New tool highlights activity of key cellular signal

ell, the nucleus and the mitochondria.

Because cyclic AMP does so many things, understanding how it works has broad implications, says Zhang. For example, the main enzyme that degrades cyclic AMP is being targeted by drug companies for potential application in inflammation, allergy, asthma or chronic lung disease, conditions that may be helped by increasing cyclic AMP. More recently, altered regulation of cyclic AMP has been tied to the risk of stroke occurrence and to heart conditions including dilated cardiomyopathy.

Zhang says her lab will use the new and other fluorescent probes to study the role of cyclic AMP in smooth muscle cells, such as those in blood vessels, in inflammation, and in fat cells, where it may be involved in diabetes-related processes.


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Contact: Joanna Downer
jdowner1@jhmi.edu
410-614-5105
Johns Hopkins Medical Institutions
15-Nov-2004


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