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Antidepressants decrease chronic pain by inhibiting TNF production in brain, UB researchers find

These results will provide insight into diseases, including chronic pain, caused by alterations in the levels of TNF or by changes in the regulation of neurotransmitter release from neurons," Spengler said.

"Knowledge obtained from these experiments will provide a better understanding of how neurons produce and regulate the production of specific proteins that ultimately control neurotransmission. While these studies establish a role for TNF in the treatment of depression, as well as for chronic pain, our next investigations, already on-going, will identify the precise mechanism of action for the analgesic effect and for the delayed clinical effect of antidepressant drugs," he said.


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Contact: Lois Baker
ljbaker@buffalo.edu
716-645-5000 x1417
University at Buffalo
10-Nov-2003


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