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Brain synapse formation linked to proteins, Stanford study finds

uld potentially turn on synapse formation," Barres said.

People with epilepsy face the opposite problem. In this disease, the area of the brain where the epileptic attack originates contains both a mass of glial cells and neurons with excessive synapses. Barres suggested that the glial scar might have originally triggered the additional synapses to form. Learning more about how glia trigger synapse formation could help prevent the disabling condition.

Co-first authors on this paper were Karen Christopherson, PhD, postdoctoral fellow, and Erik Ullian, PhD, a postdoctoral fellow now at UCSF. Other Stanford researchers who contributed to this work are undergraduate students Caleb Stokes and Christine Mullowney.


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Contact: Amy Adams
amyadams@stanford.edu
650-723-3900
Stanford University Medical Center
10-Feb-2005


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