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Inflammatory pain may help deter inflammatory diseases like arthritis

in molecules have been shed from neutrophils, the patients' poor condition could be explained in part by the fact that neutrophils continued to accumulate at the site of injury, prolonging the inflammatory response, said Strausbaugh.

"We're very excited about the direction of our research," said Strausbaugh. "Our finding provides quantifiable evidence that the pain circuits are involved in controlling inflammation."

Other co-authors of the study included Paul G. Green, PhD, an adjunct assistant professor of oral and maxillofacial surgery, Ernest Lo, MS, a student, David B. Reichling, PhD, an assistant research physiologist at the NIH Pain Center at UCSF, Kirsten Tangemann, PhD, a postdoctoral fellow in the Department of Anatomy and Program in Immunology at UCSF, and Steven D. Rosen, PhD, a professor, in the Program in Biomedical Sciences and the Department of Anatomy and Program in Immunology at UCSF.

The UCSF study was funded by the National Institutes of Health.


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Contact: Jennifer O'Brien
jobrien@pubaff.ucsf.edu
415-476-2557
University of California - San Francisco
30-Aug-1999


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