entral branch," he adds. "The problem was not that the adult central nervous system is hostile territory for growth, as previously thought. The problem is getting the injured cells to grow. Now we need to identify the molecular signals that induce this growth and the genes on which they act. If we can find ways to turn those signals on without the peripheral nerve injury and apply them soon after patients suffer spinal cord injury, we may finally achieve what was once seen as an unreachable goal: reconnection of a severed spinal cord."

The study was supported by a grant from the International Spinal Research Trust.

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Contact: Susan McGreevey
smcgreevey@partners.org
617-724-2764
Massachusetts General Hospital
28-May-1999

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