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Other highlights of the May 21 JNCI

  • Selenium May Lower Risk of Esophageal Cancer in People with Barrett's Esophagus: Higher serum selenium levels appear to be associated with a reduced risk of progression toward esophageal cancer among people with Barrett's esophagus, according to a new study. Rebecca E. Rudolph, M.D., of the Fred Hutchinson Cancer Research Center, Seattle, and her colleagues examined 399 people with Barrett's esophagus and found that people with higher concentrations of selenium in their blood were less likely to have increased 4N fraction, aneuploidy, and high-grade dysplasia-three markers of cancer progression. These individuals also were less likely to have lost the portion of chromosome 17 that contains the p53 tumor suppressor gene. The authors suggest that "selenium might reduce cancer risk among persons with Barrett's esophagus by preventing the inactivation of p53 or by preventing further neoplastic progression after p53 has been inactivated."

    Contact: Kristen Woodward, Fred Hutchinson Cancer Research Center, 206-667-5095; kwoodwar@fhcrc.org.

  • Cyclin D1 May be Involved in Ovarian Cancer Growth: Researchers have found a second mechanism through which lysophosphatidic acid (LPA) may promote ovarian tumor growth. Yu-Long Hu, Ph.D., and Robert Jaffe, M.D., of the University of California, San Francisco, and their colleagues found that LPA treatment increases cyclin D1 protein levels in ovarian cancer cells but not in normal ovarian cells. Cyclin D1 is a key cell cycle regulator that is overexpressed in various human cancers, including ovarian cancer. A previous study found that LPA can stimulate ovarian cancer growth by increasing the expression of vascular endothelial growth factor (VEGF), which promotes angiogenesis (the formation of new blood vessels). The new findings suggest that LPA stimulates ovarian tumor growth through dual mechanisms. Finding ways to block these pathways may l
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Contact: Linda Wang
jncimedia@oupjournals.org
301-841-1287
Journal of the National Cancer Institute
20-May-2003


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