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Type 1 diabetes safely arrested with short-term use of new drug

by fusing certain parts of a mouse antibody with that of a human antibody to retain the desired binding specificity without provoking an immune response as a mouse antibody would.

It is less toxic than cyclosporine and other earlier immunosuppressive drugs because it is administered for only two weeks. Briefer immunosuppression prevents the side effects which were severe in some cases with the earlier drugs including infections and cancer. Moreover, the drug is more specific and does not cause other organ toxicity such as kidney disease.

It is less toxic than other OKT3 monoclonal antibody drugs because it doesnt activate aggressive inflammatory cytokines of the immune system, which have led to fever, nausea, headache, hypertension and even cardiac problems. This drug was developed by mutating a portion of the antibody so that it does not activate T cells thus avoiding the release of the cytokines.


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Contact: Annie Bayne
as862@columbia.edu
212-305-3900
Columbia University Medical Center
29-May-2002


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