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Unexpected target may unlock the door to fighting sepsis

ed, high levels of C5a generated, which bind to the circulating neutrophils and cause their de-activation. They then lose ability to kill bacteria because their ability to generate hydrogen peroxide is suppressed."

Ward says the next steps are for these findings to be verified in other models of sepsis and in other non-human animals. Clinically, he says, the first thing that should be done is human trials that do not involve interventions. This would include taking blood samples, looking for C5a presence on neutrophils and at what point they peak, and trying to determine whether neutrophils in human sepsis patients are defective in their ability to produce hydrogen peroxide.


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Contact: Peter Barkey
pbarkey@umich.edu
734-764-2220
University of Michigan
29-Jun-1999


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