An article published in the Journal of Thrombosis and Haemostasis looks specifically at airborne particulate matter resulting mainly from the combustion of fuel, including coal and also from forest fires. Evidence shows that both short- and long-term exposure to these particulates is associated with death from cardiovascular and respiratory illnesses, and more specifically from myocardial infarction.

Additionally, this research, based on a previous study, reveals that those patients with damaged arteries are most at risk to suffer from lung inflammation and fatal blood clots.

Each year, 1.1 million people experience myocardial infarction, which results from the obstruction of a diseased coronary artery.

This study is published in the Journal of Thrombosis and Haemostasis. Media wishing to receive a PDF of the article, please contact medicalnews@bos.blackwellpublishing.net. Also available are two letters to the editor (Effects of ambient ozone on the procoagulant status and systemic inflammatory response by Hermans et al and Exposure to diesel exhaust nanoparticles does not induce blood hypercoagulability in an at-risk population by Blomberg et al.) which challenge these results. They are also available to media upon request.

Anyone sensitive to the issue and willing to contribute to help investigating further into the mechanisms involved with thrombosis and haemostasis associated with air pollution are welcome to send their comments to the Editor-in-Chief of Journal of Thrombosis and Haemostasis at editor@journalth.com. All submissions will be evaluated for publication in a special JTH Forum.

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Contact: Sharon Agsalda
medicalnews@bos.blackwellpublishing.net
781-388-8507
Blackwell Publishing Ltd.
26-Sep-2005