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Hopkins study proves cochlear implants prevent or reverse damage to brain's auditory nerve system

New research at Johns Hopkins has clearly demonstrated the ability of cochlear implants in very young animals to forge normal nerve fibers that transmit sound and to restore hearing by reversing or preventing damage to the brain's auditory system.

The findings in cats, published in Science online Dec. 2, help explain why implants are up to 80 percent successful in restoring hearing in young children born deaf, but rarely effective when implanted in congenitally deaf adults, the researchers say.

"What we think this study tells parents of deaf children is that if cochlear implants are being considered, the earlier they're done the better," says David Ryugo, Ph.D., the lead investigator in the study. "There is an optimal time window for implants if they are to avoid permanent rewiring of hearing stations in the brain and the long-term effects on language learning that can result," adds Ryugo, a professor of otolaryngology and neuroscience at The Johns Hopkins University School of Medicine and its Hearing and Balance Center.

The Hopkins team, building on years of experience with cochlear implants in children and adults, now has more evidence to support their recommendation that the devices be installed by age 2, or earlier. More than 10,000 children are born deaf each year in the United States, and an estimated 1.5 million people are believed to be good candidates for cochlear implants.

Between ages 1 and 2, children's skulls are almost fully grown, Ryugo notes, minimizing complications from brain surgery and greatly reducing the risk that the electrical wiring will loosen or pull away from their attachments under the scalp.

Cochlear implants are tiny devices designed to mimic the work of a snail-like structure in the inner ear containing fluid-filled canals and tissues. One of these is the organ of Corti, which detects pressure impulses and initiates electrical signals that travel along the inner ear's auditory nerve to the
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Contact: David March
dmarch1@jhmi.edu
410-955-1534
Johns Hopkins Medical Institutions
1-Dec-2005


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