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JCI Table of Contents, April 3, 2006

d survive demonstrated higher rates of myocyte death as well as elevated fibrosis, or scarring, of the heart tissue. The study suggests that alpha1A/B and Gq signaling are specifically necessary for a normal response to increased pressure on the heart, and is part of a molecular signaling program used by the heart to adapt to stress.

In an accompanying commentary, Stephen B. Liggett from the University of Maryland School of Medicine in Baltimore notes that, "although previous studies have suggested that approaches that decrease this signaling might be protective against the development of heart failure or be beneficial in treatment, the data obtained from this study of alpha1A/B knockout mice provide support for the potential deleterious effects of alpha1-adrenergic blockade."

TITLE: alpha1-Adrenergic receptors prevent a maladaptive cardiac response to pressure overload

AUTHOR CONTACT: Paul C. Simpson
San Francisco VA Medical Center, San Francisco, California, USA

Phone: (415) 221-4810 ext. 3200; Fax: (415) 379-5570; E-mail: paul.simpson@ucsf.edu

View the PDF of this article at: https://www.the-jci.org/article.php?id=22811

ACCOMPANYING COMMENTARY:

TITLE: Cardiac 7-transmembrane-spanning domain receptor portfolios: diversify, diversify, diversify

AUTHOR CONTACT: Stephen B. Liggett
University of Maryland School of Medicine, Baltimore, Maryland, USA

Phone: (410) 706-6256; Fax: (410) 706-6262; E-mail: sligg001@umaryland.edu

View the PDF of this article at: https://www.the-jci.org/article.php?id=28234

CARDIOLOGY

The heart teams with the kidney to control blood pressure

To control blood pressure, the kidney has evolved a m
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Contact: Brooke Grindlinger
press_releases@the-jci.org
212-342-9006
Journal of Clinical Investigation
3-Apr-2006


Page: 1 2 3 4 5 6 7 8 9 10 11 12

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