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Preventing spinal cord injury during aortic surgery

oxygen species, oxidative DNA damage, and cell death while having no effect on expression of DNA repair enzymes.

These studies demonstrate the importance of mitochondria in release of reactive oxygen species and in initiation of cell death pathways during ischemia-reperfusion injury. In particular, the establishment of a correlation between the structural integrity and the function of mitochondria in ischemia-reperfusion injury is shown for the first time in an animal model of spinal cord injury.

Dr. Wei and colleagues are optimistic of the impact that their results hold for improving the outcome of aortic surgery. Combined with future research on ischemia-reperfusion spinal injury, diazoxide may further decrease the likelihood of neurological deficits following aortic aneurysm surgery.


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Contact: Audra Cox
acox@asip.org
301-634-7409
American Journal of Pathology
21-Jun-2006


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