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Study implicates protein as a trigger of advanced prostate cancer recurrence

st a portion of these tumor recurrences. Nupams study nails down the mechanism by which that conversion happens."

Among the experiments were those that involved mice that were unable to produce androgen. The animals were implanted with human prostate tumor cells containing an activated form of Ack1. "We found that when prostate tumor cells express activated Ack1, cancer grew aggressively in these mice," Mahajan said. "This mimics what happens in patients undergoing hormone therapy."

The researchers noted that approximately one-third of androgen-independent human prostate tumors contain an activated Ack1 molecule. "The study is telling us this is a target for therapy and perhaps a very important target for therapy," Earp said.


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Contact: L. H. Lang
llang@med.unc.edu
919-843-9687
University of North Carolina School of Medicine
8-May-2007


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