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Treatment of in-hospital cardiac patients is focus of Jefferson University presentations

searchers found that acute wound healing was significantly delayed in the mice lacking Fibulin-2. After 72 hours, they found that the damaged (infarct) area was much larger in the mice lacking Fibulin-2.

The researchers found that the wound healing process was much slower in the mice lacking Fibulin-2 after a heart attack due to delayed scar tissue formation in the damaged area. The study demonstrated that Fibulin-2 facilitates heart wound healing probably with the help of (TGF)-?, a growth factor that promotes wound healing.

The researchers are also currently looking into the role of Fibulin-2 as a factor in the development of chronic heart failure.

Members of the team who conducted this study at Thomas Jefferson University in Philadelphia, Pa., and Alfred I DuPont Hospital for Children in Wilmington, Del., are:

Takeshi Tsuda, M.D.;

Erhe Gao, M.D., Ph.D.;

Francois X. Sicot, Ph.D.;

Hailong Dong, M.D., Ph.D.;

Dessislava Markova, Ph.D.,

Xinliang Ma, M.D., Ph.D. and

Mon-Li Chu, Ph.D.


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Contact: Nan Myers
nan.myers@jefferson.edu
215-955-6300
Thomas Jefferson University
27-Mar-2007


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